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Because ferroptosis relies heavily on cellular lipid, amino acid, and iron metabolic states, growing evidence indicates that effective prevention or intervention for ferroptosis in OA demands an in-depth understanding and modulation of these reprogrammed metabolic processes so as to curb OA progression at its source
15, Issue 2, Article 66).
Age and GSH metabolism in rat cerebral cortex, as related to oxidative and energy parameters
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The improvement in the frailty status was less compared to L-carnitine group
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